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Mutants of the thick veins (tkv) gene have been described previously (as slater alleles) as embryonic lethal, lacking dorsal epidermis, but not as showing a recognizable dorsoventral phenotype. The maternal alteration of function coupled with zygotic reduction of function of tkv is strongly ventralizing. In double heterozygous combinations in the mother, tkv mutations increase the ventralizing effect of dominant, weakly ventralizing alleles of the maternal effect, dorsoventral genes easter and cactus.
Receptor protein serine/threonine kinase; tkv
200 µL
200 µL
200 µL
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